Intracerebral hemorrhage (ICH) is the type of stroke with the highest mortality and disability. There is still no effective treatment. The scarcity of animal models that strictly simulate the clinical characteristics of ICH may be the root cause for hindering the research of ICH. This article reviews the existing spontaneous ICH mouse models and found two hypertensive ICH models，one spontaneous deep ICH model and five cerebral amyloid angiopathy models. One of the hypertensive models was double transgenic mice，which overexpressed human renin and angiotensinogen，leading to hypertension in mice. However，the occurrence of this model of ICH required a combination of a high-salt diet and a nitric oxide synthase inhibitor. The other hypertensive model utilized slow subcutaneous release of angiotensin Ⅱ and inhibition of nitric oxide synthase to cause chronic hypertension，followed by acute injection of angiotensin Ⅱ to further increase blood pressure and cause dramatic fluctuations in blood pressure to induce ICH. The spontaneous deep ICH model may be the most ideal mouse model for studying ICH except for hypertension. In addition，the five cerebral amyloid angiopathy models were all established in transgenic mice with amyloid precursor protein mutations，but the research on the characteristics of ICH was relatively scarce. At present，spontaneous ICH still lacks widely recognized and used animal models. The ideal ICH model should be able to simulate the development characteristics of the disease to the greatest extent. Only by carrying out ICH basic research that meets clinical needs can it be expected to provide new and effective targets on ICH prevention and treatment.